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Management of emergencies and cardiopulmonary resuscitation
Airway
and Breathing
Cardiac Arrest and Inadequate Circulation
CARDIAC
ARREST AND INADEQUATE CIRCULATION
Cardiac arrest exists when there is no detectable heartbeat,
major pulse or other sign of cardiac output; the patient is
completely unresponsive and breathing stops within a few seconds.
Unlike breathing, it is less obvious when there is no blood
circulation, especially in patients with dark skins.
You must look for and make the specific diagnosis of circulatory
arrest:
| :: |
Feel
for a pulse in the carotid or femoral artery |
| :: |
Feel
for an apex heartbeat |
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Listen
at the apex with a stethoscope |
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Look
for cyanosis or pallor in the tongue. |
Having
made the diagnosis, the immediate first step must be to
do external cardiac massage (ECM). This must start immediately
you decide that there is no circulation. Do not hesitate
to start ECM if you cannot detect a heartbeat. The action
of ECM will alert others more effectively than simply saying “There’s
a cardiac arrest”. It is best to do both.
In cardiac arrest, keep ventilating and continue ECM until
there is a response or you decide to stop treatment.
ECM should be performed while positioned well above the patient
(Figure 13.1).
At this stage, you are temporarily averting the fatal consequences
of cardiopulmonary arrest. The ABC routine is life saving,
but only for a few minutes. Some other treatment must be given
and normal circulation must be restored if the patient is to
survive.
Two people should be assigned duties: ventilation and ECM.
They will need relief when they get tired. Assign a third person
to feel for the femoral pulse and report to you if it returns.
Your next priority is to diagnose the problem with the circulation
and correct it. Your action to achieve this will depend on
the facilities you have. You may have no electronic monitoring
devices or you may have an ECG and perhaps a defibrillator.
Very often, a device is present in the hospital but does
not work when needed or it has to be brought from somewhere
else, causing delay. It may be locked in someone’s
office. You may have an ECG monitor, but have no chest
electrodes, or they have dried up through age or are of the
wrong type. The mains power lead may have been lost or stolen.
The hospital generator may have to be switched on.
There may, therefore, be many reasons why you think that you
have ECG diagnosis at your hospital whereas in fact you do
not. Check now if, in fact, an ECG monitor is available and
if it works when connected to a patient. Remind yourself how
to connect it up.
Do not waste time during a cardiac arrest trying to make an
ECG machine work.
When there is no ECG diagnosis
| 1 |
Give
a chest thump: this is a single blow with the closed
fist over the sternum, only done early in a witnessed
cardiac arrest, to try and jolt the heart into action. |
| 2 |
Give
epinephrine (adrenaline) 1 mg intravenously. |
| 3 |
Continue
CPR. Pause in CPR every minute or two to feel for pulsations
and listen for the heartbeat. If absent, continue CPR. |
| 4 |
Give
atropine 1 mg followed by 2 more doses of epinephrine
1 mg. Effective ECM will carry the epinephrine round
into the ventricles and coronary arteries where it
will have its effect. |
| 5 |
During
this time, insert an intravenous cannula and start
infusion, as below. |
Epinephrine (adrenaline) is life saving in many cases of cardiac
arrest. Always use it once the diagnosis is made, even if you
do not know the cause of the arrest.
It is usual to abandon CPR if there is no response after
20–30
minutes or if three doses of epinephrine have not produced
signs of a heartbeat.
When there is an ECG diagnosis
As you start ECM, call for the ECG monitor. In cardiac arrest,
there are three key ECG appearances at cardiac arrest:

Unless you are familiar with the normal sinus rhythm trace
(and the benign arrhythmias which do not require immediate
treatment), you will not be able to make a decision based on
what is shown there.
In order of frequency of occurrence in countries where ischaemic
heart disease is rare, the cardiac arrest rhythms are as follows.
Asystole
You will see a straight or smooth wavy line. You may see occasional
widened complexes, but no pulse can be felt in the femoral
artery. There should always be some electrical activity when
ECM is being carried out. A steady straight line may mean the
machine is not connected.
Asystole is the terminal event in many severe illnesses, but
may be acutely caused by:
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Septicaemia |
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Hypoxia |
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Excessive
vagal tone |
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Electrolyte
abnormalities |
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Severe
hypotension. |
Treat with epinephrine as above and atropine. The prognosis
is very poor.
Pulseless electrical activity (sinus rhythm)
This is also called electromechanical dissociation (EMD). There
is a near-normal ECG pattern, but no detectable pulse. There
are many causes of this situation and, in the heat of the moment,
you have to think clearly. Some of the important causes are:
| • |
Overdose
of anaesthetic agent |
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Hypovolaemia/blood
loss |
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Hypoxia
(or other ventilation problem) |
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Septic
or other toxaemia |
| • |
Pulmonary
embolus |
| • |
Cardiac
tamponade |
| • |
Tension
pneumothorax |
| • |
Hypothermia. |
For
the anaesthetist, the first three are the most common.
To treat pulseless electrical activity
Look for a cause. Give epinephrine, if needed.
A glance at the list above should tell you that some of the
causes are reversible without drug treatment and that this
may not be a true cardiac arrest. If you can withdraw or correct
the cause of the arrest (by switching off halothane, increasing
intravenous fluids, correcting a problem with the anaesthesia
circuit), this will be safer than giving an intravenous bolus
of epinephrine.
If there is no detectable circulation after two or three minutes
of CPR, even with a diagnosis and corrective measures, give
epinephrine as for asystole. The prognosis is good, if the
cause can be found.
Ventricular fibrillation
A coarse or fine jagged line denotes chaotic ventricular activity
(Figure 13.3). A defibrillator is required.
To treat, defibrillate. This needs training and experience.
|
| 1 |
Start
with 2 x 200 joules DC shock (2 joules/kg body weight)
followed by 360 joules if sinus rhythm does not return
promptly. |
| 2 |
Put
electrode jelly on the chest, check the orientation of
the paddles (labelled “apex” and “sternum”)
and press them firmly on the chest. |
| 3 |
Tell
everyone to stand clear and shock across the heart. No
one should be touching the patient or anything that is
touching the patient, including the resuscitation bag,
as most things conduct electricity. |
If you have no defibrillator, a chest thump or epinephrine
may produce sinus rhythm.
Prognosis is good, especially if the precipitating cause was
halothane and epinephrine interaction or hypoxia.
Haemorrhage
External bleeding can be controlled, usually with pressure.
Bleeding into body cavities may be apparent only later; for
example, when the circulation has been restored and the rise
in blood pressure causes more bleeding and a second fall in
blood pressure.
Shock
Shock is a pathological, life threatening condition in which
the oxygen supply to the tissues of the body fails. The cause
is usually one of the following:
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Hypovolaemia
(bleeding) |
| • |
Sepsis |
| • |
Acute
anaphylaxis: from allergy or drug reaction |
| • |
Neurogenic
(after spinal trauma) |
| • |
Heart
failure (left ventricular failure). |
There may be more than one cause of shock. In surgical patients,
look for hypovolaemia and sepsis first.
| • |
In
hypovolaemic shock, the circulating volume is reduced
by loss of blood or other fluid (e.g. burn transudate).
Rapid fluid replacement, starting with normal saline
or Hartmann’s solution, should restore the circulation
towards normal. |
| • |
In
septic shock, the circulating volume may be normal, but
blood pressure is low and tissue circulation is inadequate.
Support the circulation with volume infusion, but it
may not respond as in hypovolaemic shock. |
| • |
In
acute anaphylaxis, give epinephrine and intravenous fluids. |
| • |
Neurogenic
shock follows large neurological injuries: e.g. spinal
cord damage. The heart rate is often low and atropine
and fluids will be helpful. |
Heart
failure is beyond the scope of this book. The prognosis is
poor when it occurs intra- or postoperatively. Fluids will
not help as the circulation is overloaded.
Unconsciousness
Unconsciousness may have many causes including:
| • |
Head
injury |
| • |
Hypoglycaemia |
| • |
Ketoacidosis |
| • |
Cerebrovascular
event |
| • |
Hypoxia |
| • |
Hypotension |
| • |
Hypertension
and eclampsia |
| • |
HIV
infection |
| • |
Drug
intoxication. |
Assess
the response to stimuli, look at the pupils initially and
re-examine them later to follow progress. Look for unequal
pupils or other localizing signs that may show intracranial
haematoma developing.
In many instances, you may attend to and stabilize other systems
first and await the return of consciousness as cerebral perfusion
and oxygenation improves. After cardiac arrest, a patient who
initially had fixed dilated pupils may show smaller pupils
after effective CPR. This indicates that a favourable outcome
may be possible.
The supine unconscious patient with a full stomach is at grave
risk of regurgitation and aspiration due to the unprotected
airway. However, if a comatose patient has a clear airway and
vital signs are normal:
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Avoid
intubation as this will involve drug administration and
complicate the subsequent diagnosis |
| • |
Nurse
the patient in the recovery position |
| • |
Monitor
the airway and await progress and diagnosis (Figure
13.4). |
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During CPR, ask yourself: is the patient responding? If not,
why not?
Airway
and Breathing
Cardiac Arrest and Inadequate Circulation

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Recognize shock by:
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Tachycardia (may be the only sign in
a child)
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Thready pulse
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Narrow pulse pressure: e.g. 110/70
becomes 95/75
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Cold hands and feet
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Sweating, anxious patient
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Breathlessness and hyperventilation
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Confusion leading to unconsciousness.
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